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Antibodies against glutamic acid decarboxylase (GAD) are a hallmark of type 1 diabetes and its late-onset variant, latent autoimmune diabetes of the adult (LADA).1 While cerebrovascular disease and dementia are common causes of cognitive dysfunction and neurological deficits in diabetic patients,2,3 rare autoimmune disorders of the central nervous system such as stiff-person syndrom or limbic encephalits can also occur on the basis of shared GAD autoimmunity.4,5. This article is protected by copyright. All rights reserved.

Citation

Stoyan Popkirov, Seena Sebastian, Fatme Seval Ismail, Jörg Wellmer. Latent autoimmune diabetes and limbic encephalitis with antibodies against glutamic acid decarboxylase. Journal of diabetes. 2017 Nov 20


PMID: 29154389

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