QuickView for dichlorphenamide (compound)

Dichlorphenamide (USP); Diclofenamida [INN-Spanish]; Diclofenamide (JP15); CAS-120-97-8; m-Benzenedisulfonamide, 4,5-dichloro-; 4,5-Dichloro-m-benzenedisulfonamide; 4-11-00-00555 (Beilstein Handbook Reference); Dichlorophenamide; Diclofenamidum [INN-Latin]; 3,4-Dichloro-5-sulfamylbenzenesulfonamide.
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Dichlorphenamide (USP); Diclofenamida [INN-Spanish]; Diclofenamide (JP15); CAS-120-97-8; m-Benzenedisulfonamide, 4,5-dichloro-; 4,5-Dichloro-m-benzenedisulfonamide; 4-11-00-00555 (Beilstein Handbook Reference); Dichlorophenamide; Diclofenamidum [INN-Latin]; 3,4-Dichloro-5-sulfamylbenzenesulfonamide; Dichlorphenamide; Glaucol; Prestwick0_000809; Prestwick1_000809; Daranide; 1,3-Disulfamyl-4,5-dichlorobenzene; BRN 2703329; D00518; C07459; EINECS 204-440-6; 4,5-Dichloro-1,3-disulfamoylbenzene; NCGC00016371-01; Prestwick_1071; 1,3-Disulfamoyl-4,5-dichlorobenzene; CB 8000; 120-97-8; Dichlorphenamid; Dichlofenamide; Antidrasi; HSDB 3267; Daranide (TN); 1,3-Benzenedisulfonamide, 4,5-dichloro-; Oratrol; Dichlorphenamide [BAN]; SPBio_002598; Diclofenamide
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Diclofenamidum [INN-Latin]; Dichlorophenamide; Diclofenamida [INN-Spanish]; 4,5-Dicholorobenzene-1,3-disulfonamide; Dichlorphenamid; Dichlofenamide; Diclofenamide

Dichlorphenamide is an oral carbonic anhydrase inhibitor indicated for adjunctive treatment of: chronic simple (open-angle) glaucoma, secondary glaucoma, and preoperatively in acute angle-closure glaucoma where delay of surgery is desired in order to lower intraocular pressure. Carbonic anhydrase inhibitors reduce intraocular pressure by partially ...
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Dichlorphenamide is an oral carbonic anhydrase inhibitor indicated for adjunctive treatment of: chronic simple (open-angle) glaucoma, secondary glaucoma, and preoperatively in acute angle-closure glaucoma where delay of surgery is desired in order to lower intraocular pressure. Carbonic anhydrase inhibitors reduce intraocular pressure by partially suppressing the secretion of aqueous humor (inflow).
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Carbonic anhydrase inhibitors reduce intraocular pressure by partially suppressing the secretion of aqueous humor (inflow), although the mechanism by which they do this is not fully understood. Evidence suggests that HCO^3- ions are produced in the ciliary body by hydration of carbon dioxide under the influence of carbonic anhydrase and d...
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Carbonic anhydrase inhibitors reduce intraocular pressure by partially suppressing the secretion of aqueous humor (inflow), although the mechanism by which they do this is not fully understood. Evidence suggests that HCO^3- ions are produced in the ciliary body by hydration of carbon dioxide under the influence of carbonic anhydrase and diffuse into the posterior chamber which contains more Na⁺ and HCO^3- ions than does plasma and consequently is hypertonic. Water is then attracted to the posterior chamber by osmosis, resulting in a drop in pressure.
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