BaseSpace
Correlation
Engine-Public
Sign In
Register
Correlation Engine 2.0
Home
My Data
Bookmarks
Collaborations
Inbox
Import Your Data
QuickView
FAQ
What is QuickView?
What can my QuickView results tell me?
What are the sources for the General Info tab in QuickView?
More QuickView FAQs
Back to top
QuickView
Curated
Studies
Body
Atlas
Disease
Atlas
Pharmaco
Atlas
Knockdown
Atlas
Genetic
Markers
Pathway
Enrichment
Literature
Clinical
Trials
0
Meta-
Analysis
QuickView
Search sequence regions
(e.g.
Metabolism of nitric oxide
,
Acetaminophen
,
rs4950928
,
FABP1
,
Influenza
,
Prostate
)
Organisms
Chromosomes
Start
Stop
Homo Sapiens
Mus Musculus
Rattus Norvegicus
C. Elegans
D. Melanogaster
Saccharomyces Cerevisiae
QuickView
Go back to main search
Bookmark
Forward
QuickView
for
hyperaldosteronism
(phenotype)
Summary
General Info
Genetic Markers
Most Correlated Genes
Most Correlated SNPs
CLCNKB
BSND
SCNN1B
SCNN1G
CLCNKA
No SNPs found
Explore Genetic Markers Results
Pathway Enrichment
Most Correlated Biogroups
sodium ion transmembrane transport
excretion
Epithelial sodium channel, chordates
AR_02
Epithelial sodium channel, conserved site
Explore Pathway Enrichment Results
Curated Studies
Most Correlated Studies
Adenomas of hyperaldosteronism patients with mutations in KCNJ5, CACN1D, or ATP1A
OMIM - Liddle syndrome
OMIM - Metabolic disease
Adrenocortical carcinomas and adenomas with differing beta-catenin staining profiles
OMIM - Aldosterone disorder
Explore Curated Studies Results
Literature
Most Relevant Literature
Parathyroid hormone, a crucial mediator of pathologic cardiac remodeling in aldosteronism.
Primary aldosteronism and metabolic syndrome.
Primary aldosteronism: from bench to bedside.
The prevalence of familial hyperaldosteronism in apparently sporadic primary aldosteronism in German…
Primary aldosteronism: the role of confirmatory tests.
Explore Literature Results
Clinical Trials
Most Relevant Clinical Trials
Tissue K+ in Primary Hyperaldosteronism
HyperAldosteronism in Pregnancy Predicted Impacts (H.A.P.P.I. Trial)
Primary Hyperaldosteronism and Ischemia-reperfusion Injury
Study of Myocardial Interstitial Fibrosis in Hyperaldosteronism
Proof-of-concept for the Aldosterone Synthase Inhibitor LCI699 in Patients With Primary Hyperaldoste…
Explore Clinical Trials Results
search
→
result
search
→
result
See more about this page
See complete FAQ