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HbAS
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Epilepsy-associated SCN2A (NaV1.2) variants exhibit diverse and complex functional properties.
Inhibitory effect of atomoxetine on Nav1.2 voltage-gated sodium channel currents.
Pathogenic SCN2A variants cause early-stage dysfunction in patient-derived neurons.
A mutation in the neonatal isoform of SCN2A causes neonatal-onset epilepsy.
Functional correlates of clinical phenotype and severity in recurrent SCN2A variants.
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The Value of HBA in the Evaluation of Idiopathic Infertility
Healthy Beat Accupunch (HBA) Exercise Program for Community Older Adults
Development and Evaluation of the Healthy Beat Accupunch (HBA) Exercise Program for the Community Ol…
A Retrospective Study on the Effect of HBA or HBB Genetic Defects on Early Embryonic Development
High Resolution HBA-MRI Using Deep Learning Reconstruction
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