QuickView for SETX;jsessionid=9373B39A6AFD7E6F48C6DA4C3B0A72B8 (gene)

1. Sen1 and Rrm3 ensure permissive topological conditions for replication termination.
2. Sen1 is a key regulator of transcription-driven conflicts.
3. Premature ovarian ageing following heterozygous loss of Senataxin.
4. Clonally expanded CD8 T cells characterize amyotrophic lateral sclerosis-4.
5. Senataxin homologue Sen1 is required for efficient termination of RNA polymerase III transcription.

There were no clinical trials for SETX;jsessionid=9373B39A6AFD7E6F48C6DA4C3B0A72B8