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myelofibrosis
(phenotype)
Summary
General Info
Genetic Markers
Most Correlated Genes
Most Correlated SNPs
JAK2
XK
HBG1
IFI27
CA1
No SNPs found
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Pathway Enrichment
Most Correlated Biogroups
MARSON_BOUND_BY_FOXP3_STIMULATED
PILON_KLF1_TARGETS_DN
nucleoplasm
regulation of nitrogen compound metabolic process
BUYTAERT_PHOTODYNAMIC_THERAPY_STRESS_UP
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Curated Studies
Most Correlated Studies
Blood gene expression profiles of patients with chronic myeloproliferative disorders
Primary myelofibrosis CD34+ mononuclear blood cells mutant for JAK2, ASXL1, EZH2, SRSF2 or MPL
Neutrophils from myeloproliferative neoplasms and leukemia HEL cells; role of JAK2 V617F mutation
Peripheral blood CD34+ cells of patients with primary myelofibrosis
Blood from essential thrombocythemia, polycythemia vera and primary myelofibrosis subjects
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Literature
Most Relevant Literature
Myelofibrosis.
Pelabresib (CPI-0610): An Exciting Novel Drug for the Treatment of Myelofibrosis.
Evolving landscape of JAK inhibition in myelofibrosis: monotherapy and combinations.
Biological drivers of clinical phenotype in myelofibrosis.
Transcription factor 3 is dysregulated in megakaryocytes in myelofibrosis.
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Clinical Trials
Most Relevant Clinical Trials
Quantitative MRI for Myelofibrosis
Reversible Secondary Myelofibrosis or Clonal Myeloproliferative Disorder
Interferon-pegyle α2a Efficiency and Tolerance in Myelofibrosis
Clonal Architecture of ASXL1-mutated Myelofibrosis
Study of Select Combinations in Adults With Myelofibrosis
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