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Avoiding chromosome pathology when replication forks collide.
The level of origin firing inversely affects the rate of replication fork progression.
Substrate-selective repair and restart of replication forks by DNA translocases.
Impediments to replication fork movement: stabilisation, reactivation and genome instability.
Chromatin dynamics at the replication fork: there's more to life than histones.
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Testing the Addition of an Anti-cancer Drug, BAY 1895344 ATR Inhibitor, to the Chemotherapy Treatmen…
A Clinical Study of TQB3824 in Subjects With Advanced Cancer
Study of Glutamine as Prophylaxis for Irinotecan Induced Diarrhea
Evaluation of a Letter Intervention Promoting a Plant-based Diet
Study of AZD6738, DNA Damage Repair/Novel Anti-cancer Agent, in Combination With Paclitaxel, in Refr…
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