QuickView for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=24908B16693F376D4A550A8387801841;jsessionid=F0BE761898701E0C0C699E0C23F3D4F7;jsessionid=DF8DDD0CE027E58B4BADA2C64D44FB52;jsessionid=869F09EF63B9B8FF134BC58C4BABD11B;jsessionid=0DF5FBD84189A3D6D1E414FC963400FF;jsessionid=F285F4C017B5F5696DA68FA1874EC301;jsessionid=3370C9519D3DF4FA0D2DF525CBA51390;jsessionid=FE91410432B6FEB8D278580615A163AD (gene)

1. CaV3.2 (CACNA1H) in Primary Aldosteronism.
2. A Novel Somatic Mutation of CACNA1H p.V1937M in Unilateral Primary Hyperaldosteronism.
3. Targeting T-type/CaV3.2 channels for chronic pain.
4. Adiponectin receptor 1-mediated stimulation of Cav3.2 channels in trigeminal ganglion neurons induce…
5. The T-type calcium channel CaV3.2 regulates insulin secretion in the pancreatic β-cell.

There were no clinical trials for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=24908B16693F376D4A550A8387801841;jsessionid=F0BE761898701E0C0C699E0C23F3D4F7;jsessionid=DF8DDD0CE027E58B4BADA2C64D44FB52;jsessionid=869F09EF63B9B8FF134BC58C4BABD11B;jsessionid=0DF5FBD84189A3D6D1E414FC963400FF;jsessionid=F285F4C017B5F5696DA68FA1874EC301;jsessionid=3370C9519D3DF4FA0D2DF525CBA51390;jsessionid=FE91410432B6FEB8D278580615A163AD