QuickView for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=5A3B51A3490978A27F371A5519BCF993;jsessionid=47ED202603A84BBD452E78A564608854;jsessionid=2C198471D0FCC0C9D5E387668EE1136F (gene)

1. CaV3.2 (CACNA1H) in Primary Aldosteronism.
2. Involvement of interaction of Cav3.2 and nociceptive TRPA1 in pathological pain transmission.
3. A Novel Somatic Mutation of CACNA1H p.V1937M in Unilateral Primary Hyperaldosteronism.
4. Novel Scorpion Toxin ω-Buthitoxin-Hf1a Selectively Inhibits Calcium Influx via CaV3.3 and CaV3.2 and…
5. Targeting T-type/CaV3.2 channels for chronic pain.

There were no clinical trials for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=5A3B51A3490978A27F371A5519BCF993;jsessionid=47ED202603A84BBD452E78A564608854;jsessionid=2C198471D0FCC0C9D5E387668EE1136F