QuickView for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=97768875D1A852069DFFB01DE8231909;jsessionid=5AF62B14C366251FF1F3720EE484FCAB;jsessionid=70F663D828C9C18990F4B4DBED94DF06;jsessionid=7EAF1352506EEA8FDD09376BE044B3ED;jsessionid=641E9B1D18CBDB3081D2D2F4B398D5B1;jsessionid=DACCE5490015398ADA2E3F06F79C877C;jsessionid=2B1373E4C4B8C06B228076B7E257F0EF (gene)

1. CaV3.2 (CACNA1H) in Primary Aldosteronism.
2. Involvement of interaction of Cav3.2 and nociceptive TRPA1 in pathological pain transmission.
3. A Novel Somatic Mutation of CACNA1H p.V1937M in Unilateral Primary Hyperaldosteronism.
4. Novel Scorpion Toxin ω-Buthitoxin-Hf1a Selectively Inhibits Calcium Influx via CaV3.3 and CaV3.2 and…
5. Targeting T-type/CaV3.2 channels for chronic pain.

There were no clinical trials for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=97768875D1A852069DFFB01DE8231909;jsessionid=5AF62B14C366251FF1F3720EE484FCAB;jsessionid=70F663D828C9C18990F4B4DBED94DF06;jsessionid=7EAF1352506EEA8FDD09376BE044B3ED;jsessionid=641E9B1D18CBDB3081D2D2F4B398D5B1;jsessionid=DACCE5490015398ADA2E3F06F79C877C;jsessionid=2B1373E4C4B8C06B228076B7E257F0EF