QuickView for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=97768875D1A852069DFFB01DE8231909;jsessionid=5AF62B14C366251FF1F3720EE484FCAB;jsessionid=B7D265CB135EDE9D5A7C1C6A29F21F00;jsessionid=B5CB6A9F8A4B4076B7B182F9A1870C7A;jsessionid=FB46EF7EA4329CFA9BF98CF05BB9F5D3;jsessionid=AAF392A8F9AF58EDFA3B7717D974973B;jsessionid=E7AC8AE4315FDC7DCC5E27FBE7779E77 (gene)

1. CaV3.2 (CACNA1H) in Primary Aldosteronism.
2. A Novel Somatic Mutation of CACNA1H p.V1937M in Unilateral Primary Hyperaldosteronism.
3. Targeting T-type/CaV3.2 channels for chronic pain.
4. Adiponectin receptor 1-mediated stimulation of Cav3.2 channels in trigeminal ganglion neurons induce…
5. The T-type calcium channel CaV3.2 regulates insulin secretion in the pancreatic β-cell.

There were no clinical trials for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=97768875D1A852069DFFB01DE8231909;jsessionid=5AF62B14C366251FF1F3720EE484FCAB;jsessionid=B7D265CB135EDE9D5A7C1C6A29F21F00;jsessionid=B5CB6A9F8A4B4076B7B182F9A1870C7A;jsessionid=FB46EF7EA4329CFA9BF98CF05BB9F5D3;jsessionid=AAF392A8F9AF58EDFA3B7717D974973B;jsessionid=E7AC8AE4315FDC7DCC5E27FBE7779E77