QuickView for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=97768875D1A852069DFFB01DE8231909;jsessionid=5AF62B14C366251FF1F3720EE484FCAB;jsessionid=C7EC49B8ECE4B7644082EF287D22E79C;jsessionid=616FD67E83BB7B09C979F0D3BB15249B;jsessionid=A4D12E3F251A8D0823422513387091A9;jsessionid=DC439AD6A81DD181BD030BB2F40C80E8;jsessionid=39C801666ABDA9296B7F78FC7EB44109 (gene)

1. CaV3.2 (CACNA1H) in Primary Aldosteronism.
2. A Novel Somatic Mutation of CACNA1H p.V1937M in Unilateral Primary Hyperaldosteronism.
3. Targeting T-type/CaV3.2 channels for chronic pain.
4. Adiponectin receptor 1-mediated stimulation of Cav3.2 channels in trigeminal ganglion neurons induce…
5. The T-type calcium channel CaV3.2 regulates insulin secretion in the pancreatic β-cell.

There were no clinical trials for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=97768875D1A852069DFFB01DE8231909;jsessionid=5AF62B14C366251FF1F3720EE484FCAB;jsessionid=C7EC49B8ECE4B7644082EF287D22E79C;jsessionid=616FD67E83BB7B09C979F0D3BB15249B;jsessionid=A4D12E3F251A8D0823422513387091A9;jsessionid=DC439AD6A81DD181BD030BB2F40C80E8;jsessionid=39C801666ABDA9296B7F78FC7EB44109