QuickView for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=97768875D1A852069DFFB01DE8231909;jsessionid=5AF62B14C366251FF1F3720EE484FCAB;jsessionid=C7EC49B8ECE4B7644082EF287D22E79C;jsessionid=B70F3FFAE8D6F540BD46126DF0554200;jsessionid=7F169CD363B166A7D741785242D26ABE;jsessionid=B9957CE942079F452A69804FAA424AAF (gene)

1. CaV3.2 (CACNA1H) in Primary Aldosteronism.
2. Involvement of interaction of Cav3.2 and nociceptive TRPA1 in pathological pain transmission.
3. A Novel Somatic Mutation of CACNA1H p.V1937M in Unilateral Primary Hyperaldosteronism.
4. Novel Scorpion Toxin ω-Buthitoxin-Hf1a Selectively Inhibits Calcium Influx via CaV3.3 and CaV3.2 and…
5. Targeting T-type/CaV3.2 channels for chronic pain.

There were no clinical trials for CACNA1H;jsessionid=D4884D264F989B51601A1791B109E9B9;jsessionid=97768875D1A852069DFFB01DE8231909;jsessionid=5AF62B14C366251FF1F3720EE484FCAB;jsessionid=C7EC49B8ECE4B7644082EF287D22E79C;jsessionid=B70F3FFAE8D6F540BD46126DF0554200;jsessionid=7F169CD363B166A7D741785242D26ABE;jsessionid=B9957CE942079F452A69804FAA424AAF