QuickView for TNRC13;jsessionid=3D765BD25ABDC6EF1EDE0A75B9F1BD6C;jsessionid=A8AB09FA390A3AE4E0D1D6F38BF237B8;jsessionid=51586F57457820B19218871CCD1B1B0A;jsessionid=1378266A38B81A4711AC8DCC93F77B7F;jsessionid=73AC8971AD1AC6EB5D00E04011873BA7;jsessionid=C057DE3D41D9400A19D1CA708AC75D4C;jsessionid=CE2049D0E55AC0DF318C4A9DF0DF865C;jsessionid=2D454D7F5CD07B99BE8D7281BAFFBF85;jsessionid=4392441200BEE4A81D6BF1744FB4CCEF;jsessionid=60F20EA7D4372A90B56BEDBFD842F050;jsessionid=CB5B2554D31D8F3B9E052BC3E87414BD;jsessionid=89D26EBF78F99A46A7163B215907B7D4 (gene)

1. ATXN2 intermediate expansions in amyotrophic lateral sclerosis.
2. Missense mutation in ATXN2 gene (c.2860C > T) in an amyotrophic lateral sclerosis patient with aggre…
3. The RNA-binding protein and stress granule component ATAXIN-2 is expressed in mouse and human tissue…
4. Ataxin-2 gene: a powerful modulator of neurological disorders.
5. Antagonistic roles for Ataxin-2 structured and disordered domains in RNP condensation.

There were no clinical trials for TNRC13;jsessionid=3D765BD25ABDC6EF1EDE0A75B9F1BD6C;jsessionid=A8AB09FA390A3AE4E0D1D6F38BF237B8;jsessionid=51586F57457820B19218871CCD1B1B0A;jsessionid=1378266A38B81A4711AC8DCC93F77B7F;jsessionid=73AC8971AD1AC6EB5D00E04011873BA7;jsessionid=C057DE3D41D9400A19D1CA708AC75D4C;jsessionid=CE2049D0E55AC0DF318C4A9DF0DF865C;jsessionid=2D454D7F5CD07B99BE8D7281BAFFBF85;jsessionid=4392441200BEE4A81D6BF1744FB4CCEF;jsessionid=60F20EA7D4372A90B56BEDBFD842F050;jsessionid=CB5B2554D31D8F3B9E052BC3E87414BD;jsessionid=89D26EBF78F99A46A7163B215907B7D4