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double-strand break repair via nonhomologous end joining
(biogroup)
Summary
General Info
Body Atlas
Most Correlated Tissues
Thymus gland
Hematopoietic stem cell of bone marrow
Endometrium
Testes
Heart ventricle
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Disease Atlas
Most Correlated Diseases
Vesicular stomatitis Indiana virus disease
Pyloric ulcer
Poisoning by drug AND/OR medicinal substance
Injury of integument
Barrett's esophagus
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Pharmaco Atlas
Most Correlated Compounds
valrubicin
MK 1775
N-(2-aminophenyl)-4-((4-pyridin-3-ylpyrimidin-2-ylamino)methyl)benzamide
ARQ 197
SN-38
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Knockdown Atlas
Most Correlated Gene Perturbations
AURKB
FUBP3
NOP2
TLK2
RPS6
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Curated Studies
Most Correlated Studies
L1000 CMAP - Neural progenitor NPC.CAS9 cells treated with small molecule perturbagens
L1000 CMAP - Breast cancer MCF7 cells treated with small molecule perturbagens (2 of 4)
Spatio-temporal transcriptome of the human brain
L1000 CMAP - Neural progenitor NPC cells treated with small molecule perturbagens
L1000 CMAP - Acute promyelocytic leukemia HL60 cells treated with ligand perturbagens
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Literature
Most Relevant Literature
There were no matches for double-strand break repair via nonhomologous end joining
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Clinical Trials
Most Relevant Clinical Trials
There were no clinical trials for double-strand break repair via nonhomologous end joining
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