QuickView for TNRC13;jsessionid=3D765BD25ABDC6EF1EDE0A75B9F1BD6C;jsessionid=A8AB09FA390A3AE4E0D1D6F38BF237B8;jsessionid=51586F57457820B19218871CCD1B1B0A;jsessionid=1378266A38B81A4711AC8DCC93F77B7F;jsessionid=73AC8971AD1AC6EB5D00E04011873BA7;jsessionid=C057DE3D41D9400A19D1CA708AC75D4C;jsessionid=8024CB861F67EABF3542FC05E9ADF376;jsessionid=FBCDA656B35EB721DC9B9CE6DE4C30A8;jsessionid=FEE1ABE31581E66107E68EB36F868F63;jsessionid=2169FE2F4A0A302BD4E44C6D0248E704;jsessionid=A5A421F9797264174D8035C3C590EDA6;jsessionid=F0E0273AB0DCE4648EEC87C3C987A995 (gene)

1. ATXN2 intermediate expansions in amyotrophic lateral sclerosis.
2. Missense mutation in ATXN2 gene (c.2860C > T) in an amyotrophic lateral sclerosis patient with aggre…
3. The RNA-binding protein and stress granule component ATAXIN-2 is expressed in mouse and human tissue…
4. Ataxin-2 gene: a powerful modulator of neurological disorders.
5. Antagonistic roles for Ataxin-2 structured and disordered domains in RNP condensation.

There were no clinical trials for TNRC13;jsessionid=3D765BD25ABDC6EF1EDE0A75B9F1BD6C;jsessionid=A8AB09FA390A3AE4E0D1D6F38BF237B8;jsessionid=51586F57457820B19218871CCD1B1B0A;jsessionid=1378266A38B81A4711AC8DCC93F77B7F;jsessionid=73AC8971AD1AC6EB5D00E04011873BA7;jsessionid=C057DE3D41D9400A19D1CA708AC75D4C;jsessionid=8024CB861F67EABF3542FC05E9ADF376;jsessionid=FBCDA656B35EB721DC9B9CE6DE4C30A8;jsessionid=FEE1ABE31581E66107E68EB36F868F63;jsessionid=2169FE2F4A0A302BD4E44C6D0248E704;jsessionid=A5A421F9797264174D8035C3C590EDA6;jsessionid=F0E0273AB0DCE4648EEC87C3C987A995